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Su GG, Pan KH, Zhao NF, Fang SH, Yang DH, Zhou Y

10.1111/j.1471-4159.1984.tb09689.x 18 FranzM.RodriguezH.LopesC.ZuberiK.MontojoJ.BaderG

Nephron 1998

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Activating pathways for creation of NADPH has been shown to protect RPE cells from oxidative damage 74 , and our findings indicate that for developing mouse retinas with RP in which all rods have been lost a possible intervention strategy would be to stimulate synthesis of NADPH

Therefore the principal drug had the greatest role in the treatment of CAG with XJZ, the assistant drug and the complement drug alone had a lesser role, and both mainly played a role together with the principal drug (Figure 7)